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The unusual triggering of the HGF/c-MET pathway may lead to tumor development via mechanisms that are both reliant on and independent of ligands

This is all based on animal models and what we understand about underlying physiology

ROS stimulates the activity of many physiological components, including mitogen-activated protein kinases (MAPK), tyrosine kinases, Rho kinases, and transcription factors (NF-kB, AP-1, and HIF-1) and increased concentration of intracellular free Ca 2+ also inactivates the protein tyrosine phosphatases (PTP)